Hongguang Cui

The paper shows that potyvirids can naturally lose their silencing suppressor coding sequences, reducing pathogenicity and enabling long-term plant-virus coexistence through genomic deletion.

This study identifies a conserved lysine/arginine-rich motif in potyviral 6K1 protein that mediates autophagy-mediated self-degradation, essential for viral infection.

The paper redefines programmed transcriptional slippage (PTS) in RNA viruses by showing it occurs more frequently and has broader implications than previously understood.

This paper develops a VIGS vector in passion fruit to study gene function in biosynthesis of health-promoting compounds.

This study identifies HCPro2 as a potyvirus-encoded scaffold protein that interacts with viral and host factors to assemble an intercellular movement complex, promoting viral infection.