William R. Miller

This study identifies FptA inactivation as a key step in cefiderocol resistance in Pseudomonas aeruginosa PA14, revealing resistance mechanisms and fitness costs.

The paper characterizes recalcitrant enterococcal bacteremia through clinical and genomic analysis, finding species-specific genetic adaptations in persistent infections.

This study shows that the MadR regulon can be activated independently of MadS through cross-talk with LiaS, enhancing Enterococcus faecalis resistance to daptomycin.

This study examines TBDR mutations in P. aeruginosa clinical isolates and their association with cefiderocol resistance and heteroresistance.

This study identifies the MadRS system and its operon madEFG as mediators of resistance to antimicrobial peptides and antibiotics in Enterococcus faecalis, linking membrane lipid changes to stress response and virulence.