Author

Ali Movasati

University of ZurichORCID 0000-0002-2178-4110Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Evolutionary Biology· 1 Genomics· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:statistical model· 2 Research Focus:variant calling· 2 Assay:whole-genome sequencing· 2 Assay:CRISPR screen· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:genome assembly· 1

Frequent Co-Authors

Karin Jutta MetzneriD· 2 shared Adriana L. HotziD· 1 shared Arne van HoeckiD· 1 shared Christine Leemann· 1 shared D. Rodrigues· 1 shared Edwin P. J. G. CuppeniD· 1 shared Francisco Martínez-JiméneziD· 1 shared Jurek Andres· 1 shared

Activity by Year

2022-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersMolecular Biology

Karin Jutta MetznerORCID

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3 papers

Recent·Most cited

Molecular BiologybioRxivPreprintJun 1, 2026

DNA Damage Response Proteins Are Involved in the Formation of Defective HIV-1 Proviruses

Koleta Michalek, Sagnik Bhattacharjee, Ali Movasati…·Karin Jutta Metzner

This study identifies DNA damage response proteins that influence the formation of defective HIV-1 proviruses through CRISPR screens and functional validation.

1 citation

humanCRISPR screen

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Evolutionary BiologyVirus EvolutionPeer-reviewedJan 1, 2026

Substantial Deceleration of Adaptation of HIV-1 Within 1,500 Generations in an Experimental Evolution: A Genomic Perspective

Ali Movasati, Christine Leemann, Kathrin Neumann…·Roland R. Regoes

This study tracks HIV-1 adaptation over 1,500 generations in human T-cell lines, finding a substantial deceleration in adaptive mutation fixation rates and fitness gains.

DOI 10.1093/ve/veag059

humanwhole-genome sequencing

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GenomicsbioRxivPreprintJun 17, 2022

Pan-cancer whole genome comparison of primary and metastatic solid tumors

Francisco Martínez-Jiménez, Ali Movasati, Sascha R. Brunner…·Arne van Hoeck

This study compares whole-genome sequences of primary and metastatic tumors across 7,152 samples, revealing cancer-type-specific genomic changes and treatment-induced evolutionary bottlenecks.

15 citations

humanwhole-genome sequencing

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