Author

Elettra Barberis

Università degli Studi del Piemonte Orientale “Amedeo Avogadro”ORCID 0000-0002-2296-9766Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:proteomics· 3 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Marcello ManfrediiD· 3 shared Armando A. GenazzaniiD· 2 shared Beatrice Pistolato· 2 shared Dmitry LimiD· 2 shared Giulia DematteisiD· 2 shared Laura TapellaiD· 2 shared Aleida La Forgia· 1 shared Alessandro CorbelliiD· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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Curved chords = mutual co-authorship

3 shared papersNeuroscience

Marcello ManfrediORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

ImmunologyCancer DiscoveryPeer-reviewedMar 11, 2025

RORγ bridges cancer-driven lipid dysmetabolism and myeloid immunosuppression

Augusto Bleve, Martina Incerti, Francesca Maria Consonni…·Antonio Sica

This study shows RORγ mediates cancer-driven lipid dysmetabolism and myeloid immunosuppression by promoting MDSC expansion and M2 TAM polarization, with cholesterol lowering reversing these effects.

28 citationstop 1%

humanproteomics

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NeuroscienceCell Death and DiseasePeer-reviewedOct 18, 2022

Protein synthesis inhibition and loss of homeostatic functions in astrocytes from an Alzheimer's disease mouse model: a role for ER-mitochondria interaction.

Laura Tapella, Giulia Dematteis, Marianna Moro…·Dmitry Lim

This study shows that in Alzheimer's disease astrocytes, protein synthesis is impaired through PERK-independent p-eIF2 activation, linked to altered ER-mitochondria interactions, affecting homeostatic functions.

34 citationstop 5%

mouseproteomics

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NeuroscienceCell CalciumPeer-reviewedSep 27, 2021

Deletion of calcineurin from astrocytes reproduces proteome signature of Alzheimer's disease and epilepsy and predisposes to seizures

Laura Tapella, Giulia Dematteis, Federico Alessandro Ruffinatti…·Dmitry Lim

This study shows that deleting calcineurin in astrocytes leads to proteome changes resembling Alzheimer's and epilepsy, and increases seizure risk in mice.

12 citations

mouseproteomics

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