Author

Catiana H. Cartwright-Acar

Fred Hutch Cancer CenterORCID 0000-0002-2682-0328Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 2 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Organism:C. elegans· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

Leslie GooiD· 2 shared Lisa M. Levoir· 2 shared Maya ContrerasiD· 2 shared Aditi AgrawaliD· 1 shared Alan M. ZahleriD· 1 shared Caleb D. MarceauiD· 1 shared Caroline KikawaiD· 1 shared Destiny R. GomeziD· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersMicrobiology

Leslie GooORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

MicrobiologyJournal of VirologyPeer-reviewedOct 8, 2024

Functional genomics screens reveal a role for TBC1D24 and SV2B in antibody-dependent enhancement of dengue virus infection

Laura Belmont, Maya Contreras, Catiana H. Cartwright-Acar…·Leslie Goo

The paper identifies TBC1D24 and SV2B as host factors required for antibody-dependent enhancement of dengue virus infection, beyond the role of FcgR.

5 citations

genetic perturbation / CRISPRhost-pathogen & immune response

𝕏 PostBluesky

MicrobiologybioRxivSep 13, 2023

The effect of single mutations in Zika virus envelope on escape from broadly neutralizing antibodies

Caroline Kikawa, Catiana H. Cartwright-Acar, Jackson B. Stuart…·Leslie Goo

This study identifies how single mutations in Zika virus envelope protein affect neutralization by broadly and narrowly specific antibodies, finding that broad antibodies are harder to escape.

DOI 10.1128/jvi.01414-23

virusproteomics

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Molecular BiologyPLoS GeneticsPeer-reviewedFeb 10, 2022

A Genetic Screen for Suppressors of Cryptic 5' Splicing in C. elegans Reveals Roles for KIN17 and PRCC in Maintaining Both 5' and 3' Splice Site Identity

Jessie M. N. G. L. Suzuki, Kenneth Osterhoudt, Catiana H. Cartwright-Acar…·Alan M. Zahler

The paper identifies KIN17 and PRCC as factors involved in maintaining 5' and 3' splice site identity during pre-mRNA splicing in C. elegans through a genetic screen and mRNA sequencing analysis.

14 citations

C. elegansbulk RNA-seq

𝕏 PostBluesky