Esteban J. Rozen

This study investigates how DDR2 affects cell mechanics and migration in neuroblastoma cells, finding that DDR2 down-regulation reduces proliferation, stiffness, and traction forces.

This study identifies DYRK3 and CAMKV as a signaling module critical for neuroblastoma cell proliferation and tumor growth, suggesting their inhibition as a potential therapeutic strategy.

The paper identifies PAK2-mediated phosphorylation of MYCN and MAX as a tumor-suppressor mechanism in neuroblastoma, with potential therapeutic strategies to restore its activity.

This study investigates how DDR2 and substrate stiffness affect cancer cell proliferation and transcriptome, revealing distinct regulatory mechanisms.