Author

Tanya N. Soliman

Queen Mary University of LondonORCID 0000-0002-4687-629XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:whole-genome sequencing· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

César SerranoiD· 2 shared David Gómez-PeregrinaiD· 2 shared Floris FoijeriD· 2 shared Molly A GuscottiD· 2 shared René WardenaariD· 2 shared Sarah Elizabeth McClellandiD· 2 shared A. GallagheriD· 1 shared Alexander Malling Andersen· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersGenomics

César SerranoORCID

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2 papers

Recent·Most cited

GenomicsbioRxivPreprintOct 22, 2026

Chromosomal instability drives continual flux in cancer genomes

Molly A Guscott, Audrey Lumeau, A. Gallagher…·Sarah Elizabeth McClelland

This study shows that chromosomal instability (CIN) drives rapid genome changes in cancer cells, generating new copy number alterations each cell cycle and increasing genomic complexity.

DOI 10.64898/2026.10.16.759545

humanwhole-genome sequencing

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Cancer BiologybioRxivPreprintSep 27, 2023

Tracking genome evolution in single cell clones reveals the rates and features of copy number alterations generated by ongoing chromosomal instability in cancer.

Molly A Guscott, David Gómez-Peregrina, Alexander Malling Andersen…·Sarah Elizabeth McClelland

The paper tracks genome evolution in single-cell clones to reveal copy number alteration rates and features in cancer driven by chromosomal instability.

3 citations

humanwhole-genome sequencing

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