Author

Maria C. Cid

Hospital Clínic de BarcelonaORCID 0000-0002-4730-0938Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 1 Research Focus:GWAS / association mapping· 1 Assay:GWAS summary statistics· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Augusto VaglioiD· 1 shared Barbara Rewerska· 1 shared Beatriz EscobariD· 1 shared Beatriz PalaciosiD· 1 shared Benjamin TerrieriD· 1 shared Chiara BaldiniiD· 1 shared Chris WallaceiD· 1 shared Covadonga Díaz-DíaziD· 1 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperGenetics

Augusto VaglioORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

PathologyCirculationPeer-reviewedJun 8, 2026

Activation of HIF2 leads to vascular remodeling and inflammation, coronary thrombosis and arterial dilation, recapitulating cardiac involvement of Kawasaki disease.

Beatriz Escobar, Ivan Menendez-Montes, Teresa Albendea-Gomez…·Silvia Martín-Puig

This study shows that activating HIF2 causes vascular remodeling, inflammation, and coronary issues similar to Kawasaki disease, using a new mouse model and human tissue analysis.

DOI 10.1161/circulationaha.125.076230

mouseimaging

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GeneticsbioRxivPreprintDec 10, 2018

Genetically distinct clinical subsets, and associations with asthma and eosinophil abundance, within Eosinophilic Granulomatosis with Polyangiitis

Paul Lyons, James E. Peters, Federico Alberici…·Kenneth G. C. Smith

This study identifies genetic subsets of EGPA associated with asthma and eosinophil levels, revealing two distinct clinical and genetic syndromes.

10 citations

humanGWAS summary statistics

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