Author

Ivan Menendez-Montes

Spanish National Centre for Cardiovascular ResearchORCID 0000-0001-5252-9502Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:metabolomics· 2 Assay:proteomics· 2 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Beatriz EscobariD· 3 shared Luis Jesús Jiménez‐BorregueroiD· 3 shared Manuel J. GomeziD· 3 shared Silvia Martı́n-PuigiD· 3 shared Alessia FerrariniiD· 2 shared Ana Vanessa Alonso· 2 shared Beatriz PalaciosiD· 2 shared Elena Bonzón‐KulichenkoiD· 2 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersDevelopmental Biology

Beatriz EscobarORCID

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3 papers

Recent·Most cited

PathologyCirculationPeer-reviewedJun 8, 2026

Activation of HIF2 leads to vascular remodeling and inflammation, coronary thrombosis and arterial dilation, recapitulating cardiac involvement of Kawasaki disease.

Beatriz Escobar, Ivan Menendez-Montes, Teresa Albendea-Gomez…·Silvia Martín-Puig

This study shows that activating HIF2 causes vascular remodeling, inflammation, and coronary issues similar to Kawasaki disease, using a new mouse model and human tissue analysis.

DOI 10.1161/circulationaha.125.076230

mouseimaging

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Developmental BiologybioRxivPreprintMay 23, 2020

Activation of amino acid metabolic program in response to impaired glycolysis in cardiac HIF1 deficient mice

Ivan Menendez-Montes, Beatriz Escobar, Manuel J. Gómez…·Silvia Martı́n-Puig

The paper investigates the effects of HIF1 deletion in cardiac progenitors, finding that it leads to impaired glycolysis and activation of an amino acid metabolic program without affecting heart development or function.

DOI 10.1101/2020.05.23.111674

mousebulk RNA-seq

𝕏 PostBluesky

Developmental BiologybioRxivPreprintMar 16, 2019

Metabolic reprogramming from glycolysis to amino acid utilization in cardiac HIF1α deficient mice.

Ivan Menendez-Montes, Beatriz Escobar, Beatriz Palacios…·Silvia Martı́n-Puig

This study investigates the effects of HIF1α deletion in cardiac progenitors during heart development, finding that HIF1 is dispensable for cardiac growth and reveals metabolic reprogramming toward amino acid utilization.

DOI 10.1101/579433

mouseproteomics

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