Matthias Hahn

Deleting 27 CDIP-candidate genes (26 confirmed CDIPs) and two phytotoxin biosynthesis genes in Botrytis cinerea nearly abolishes apple infection but leaves slow leaf lesions, pointing to redundant, unidentified host-killing factors.

The paper identifies Hip1 as a homolog of PEIE1, showing both immune signaling modulation and EDS1-dependent cell death induction in plants.

The paper identifies Gas5A, a glucanosyltransferase from Botrytis, as a cell death inducing protein in plants, showing it can trigger cell death both as a PAMP and intracellularly.

This study shows Botrytis uses actin-dependent invasive pressure and surface adherence to penetrate plant cells, reducing virulence when actin polymerization is blocked.

The paper investigates the virulence of Botrytis cinerea by generating multiple knockout mutants, revealing high redundancy in phytotoxic compounds that drive necrotrophic infection.

This study identifies retrotransposons as pathogenicity factors in the fungus Botrytis cinerea by showing their role in enhancing aggressiveness and suppressing plant defense genes through BcsRNA production.

This paper introduces a CRISPR/Cas system using ribonucleoprotein complexes and telomere vectors for efficient marker-free genome editing in Botrytis cinerea, achieving high editing rates and enabling multiple edits.