Antoine Stier

This study examines how subtle temperature changes during early life affect gut microbiome diversity and composition in wild birds, finding that rearing environment has a greater influence than genetics.

This study examines how adult king penguins respond to chronic corticosterone exposure, finding they show resistance and resilience to its negative effects on physiology.

This study examines heat stress in breeding king penguins through behavioral and physiological measurements, finding that heat stress is common and linked to reproductive failure.

The paper quantifies mitochondrial efficiency and ROS production during postnatal growth in Japanese quail, finding higher efficiency with lower ROS production.

The paper investigates CMS in Physa acuta, finding altered mitochondrial complex I in male-sterile D mitotypes and higher proton leak in K restored hermaphrodites.

This study tracks the spread and evolution of H5N1 in the sub-Antarctic and Antarctica, revealing long-distance dispersal and mammalian-adaptive mutations.

Both experimental nestbox heating (+2.83°C) and cooling (-1.72°C) during early postnatal development significantly shorten day-13 relative telomere length in wild pied flycatcher nestlings without altering body mass.

This study examines thermoregulation in moulting king penguins, finding that they experience hyperthermia and increased peripheral heat loss, supporting the warm-up hypothesis for feather development.

This study vaccinates king penguin chicks with an mRNA vaccine against H5 HPAI and finds a persistent immune response without adverse effects.

This study measures surface temperatures of captive Gentoo penguins during moult to assess thermal challenges from increased insulation and heat dissipation needs.

The paper shows mitonuclear interactions affect aerobic metabolism in bird hybrids and may explain geographic discordance between mitochondrial and nuclear genomes.

The paper shows that early-stage altricial bird embryos express genes involved in thyroid hormone pathways, suggesting they can respond to maternal thyroid hormone signals.