Author

John Jia En Chua

Agency for Science, Technology and ResearchORCID 0000-0002-5615-1014Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Adam C. PuchéiD· 1 shared Alexandros PoulopoulosiD· 1 shared Andrea J. RomanowskiiD· 1 shared Annarita PatriziiD· 1 shared Bekir AltasiD· 1 shared Cheryl BrandenburgiD· 1 shared Colin D. RobertsoniD· 1 shared Dilja Krueger‐BurgiD· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Adam C. PuchéORCID

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2 papers

Recent·Most cited

NeuroscienceBiological PsychiatryPeer-reviewedDec 26, 2023

Phosphorylation Determines Whether Neuroligin-3 is at Excitatory or Inhibitory Synapses in Different Regions of the Brain

Bekir Altas, Liam P. Tuffy, Annarita Patrizi…·Alexandros Poulopoulos

9 citations

mouseproteomics

𝕏 PostBluesky

NeuroscienceiSciencePeer-reviewedNov 20, 2023

FEZ1 participates in human embryonic brain development by modulating neuronal progenitor subpopulation specification and migration

Yinghua Qu, Jonathan Jun-Yong Lim, Ömer An…·John Jia En Chua

This study shows FEZ1 is essential for human brain development by regulating neuronal progenitor subpopulations and migration, with its deletion causing abnormalities in cortical layer formation.

6 citations

humanbulk RNA-seq

𝕏 PostBluesky