David G. Schatz

The paper demonstrates that a human antibody heavy chain variable region exon forms a stable somatic hypermutation center (SHM-C) through interaction with 3' regulatory regions, supporting AID access and SHM in non-dividing cells.

The paper determines the structure of the RAG1 N-terminal zinc-finger domain using NMR spectroscopy and identifies its evolutionary relationship with Chapaev transposases.

This study shows the SV40 enhancer targets somatic hypermutation, leading to LT truncation and potential oncogenic activity in B and kidney cells.

This study identifies ELOF1 as a critical factor for somatic hypermutation and class switch recombination by facilitating RNAPII pausing and AID targeting.