Julien Royet

This study identifies a rapid escape behavior in Drosophila larvae triggered by metabolically active pathogens, revealing multisensory detection mechanisms and their dual role in defense and pathogen spread.

The paper identifies uracil as a bacterial signal that primes Drosophila adults to respond to peptidoglycan through Duox/ROS and TrpA1/Gr66a pathways in larval neurons.

This study shows that Drosophila integrates positive and negative signals from bacterial peptidoglycan (PGN) to make behavioral decisions.

The paper shows that caffeine aversion in Drosophila requires the TrpA1-E isoform and PLC signaling in Gr66a+ neurons.

This study shows that Drosophila larvae use taste receptors to detect bacteria, which primes their immune system and improves survival after infection.

The paper shows that Drosophila bitter taste neurons use the PGRP-LB receptor and NF-κB/IMD pathway to detect bacterial peptidoglycan and influence behavior.