Author
Xiumin Chen
The University of QueenslandORCID 0000-0002-5764-6705Unclaimed ORCID profile · follow to get notified of new preprints
Biology Disciplines
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2018-2026 · peak 1/yrCo-Author Collaboration Network
10 top collaborators · 19 mutual co-author links5 papers
Recent·Most cited
- NeuroscienceJournal of Clinical InvestigationPeer-reviewed
Disrupted callosal connectivity underlies long-lasting sensory-motor deficits in an NMDA receptor antibody encephalitis mouse model
Jing Zhou, Ariele L. Greenfield, Rita P. Loudermilk…·Samuel J. Pleasure
This study shows that exposure to anti-GluN1 mAb during development causes long-term sensory-motor deficits and callosal axon changes in mice.
7 citations - NeurosciencebioRxivPreprint
CaMKII autophosphorylation but not downstream kinase activity is required for synaptic memory
Xiumin Chen, Qixu Cai, Jing Zhou…·Roger A. Nicoll
This study identifies that CaMKII autophosphorylation and binding to GluN2B are necessary for synaptic memory, while kinase activity is not required.
6 citations - NeuroscienceCell ReportsPeer-reviewed
Regulation of NMDA receptor trafficking and gating by activity-dependent CaMKIIα phosphorylation of the GluN2A subunit
Xuan Ling Hilary Yong, Lingrui Zhang, Liming Yang…·Victor Anggono
This study identifies CaMKIIα-mediated phosphorylation of GluN2A at Ser-1459 as a mechanism regulating NMDAR trafficking and gating, with implications for epilepsy.
- NeurosciencebioRxivPreprint
Phosphorylation of the alpha2 glycine receptor induces an extracellular conformational change and slows the rise and decay rates of glycinergic synaptic currents
Sharifun Islam, Xiumin Chen, Argel Estrada‐Mondragón, Joseph W. Lynch
This study shows that phosphorylation of the 2 glycine receptor at S341 induces a conformational change and alters the kinetics of glycinergic synaptic currents.
DOI 10.1101/434357