Qiyi Tang

This paper investigates how HCV remodels lipid droplets during infection, leading to mitochondrial fatty acid accumulation and metabolic activation, with implications for antiviral strategies.

This study identifies six ACE2 regions and peptides that can neutralize SARS-CoV-2, showing computational and experimental consistency across variants.

This study identifies the role of CMV viral protein IE1 in disrupting host DISC1-PML interaction, leading to neural progenitor deficits in congenital CMV infection.

The paper identifies five lethal and two live attenuated HCMV mutants by mutating pp150-capsid interactions, revealing critical residues for viral replication.

The paper identifies twelve SARS-CoV-2 proteins that induce oxidative stress, inflammation, and cell death, with ORF3a highlighted as a promising antiviral target for drug discovery.

The paper performs computational saturation mutagenesis on SARS-CoV-1 spike protein to assess stability and binding affinity, finding similar mutation effects between SARS-CoV-1 and SARS-CoV-2.

This study quantifies the effects of missense mutations on SARS-CoV-2 spike protein stability and RBD-ACE2 interaction using computational methods.