Beata Wójciak‐Stothard

This study identifies KLF6 as a key driver of endothelial dysfunction in pulmonary arterial hypertension, showing its role in promoting vascular remodelling and angioproliferative lesions.

This study develops an organ-on-chip model to investigate respiratory-vascular interactions under oxidative stress, revealing how airway injury drives vascular inflammation and remodeling in COPD.

This study identifies KLF2-induced microRNAs that protect endothelial cells and reduce pulmonary hypertension in animal models and patient samples.