Ferdi Rıdvan Kiral

This study investigates how the dyslexia-associated gene KIAA0319 regulates cell cycle during human neuroepithelium development, linking it to reading disability through effects on neuronal progenitor cells.

The paper shows that temperature-dependent changes in synaptic partner availability lead to variable connectivity and behavior in Drosophila without affecting canonical synapse ratios.

The paper shows that autophagy regulates axonal filopodia kinetics, which restricts incorrect synaptic partner choice during Drosophila brain development.

This study systematically analyzes Rab GTPase function in Drosophila neurons, revealing that some Rabs are critical for robustness to environmental challenges despite being viable in null mutants.

The paper develops a 4D analysis method and computational model to show that filopodia compete for synaptic seeding factors, leading to serial synapse formation in Drosophila R7 photoreceptor axons.