Author

Yu Pan

Chinese Academy of SciencesORCID 0000-0002-7417-4551Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Yu Pan3 papers · Harvard University

Biology Disciplines

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Physiology· 2 Neuroscience· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Approach:mechanistic / dynamical model· 1

Frequent Co-Authors

Aijing LiuiD· 2 shared Chunmiao LiuiD· 2 shared Daling ZhuiD· 2 shared He Zhu· 2 shared Xiaoxu ZhaoiD· 2 shared Yingqi WangiD· 2 shared Zhitu Bao· 2 shared Chunmu Yin· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersPhysiology

Aijing LiuORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

PhysiologybioRxivPreprintNov 11, 2026

28S-rsRNA facilitates PASMC proliferation through PKM2 β-hydroxybutyrylation

Aijing Liu, Yingqi Wang, Yu Pan…·Hang Yu

This study identifies 28S rsRNA-CTC_L20 as a regulator of PASMC proliferation in pulmonary hypertension by modulating BHB production and PKM2 β-hydroxybutyrylation.

DOI 10.64898/2026.11.09.765510

mousebulk RNA-seq

𝕏 PostBluesky

PhysiologyHypertensionPeer-reviewedJul 22, 2025

LINC00599 Promotes Pulmonary Hypertension via LLPS with G3BP1 and MYH9

Y. Wang, Lulu Yin, Shuang Zheng…·Hang Yu

This study identifies LINC00599 as a key regulator of pulmonary hypertension through m6A modification and liquid-liquid phase separation with G3BP1 and MYH9, promoting PASMC proliferation.

5 citations

mousebulk RNA-seq

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NeurosciencebioRxivPreprintJan 9, 2025

Title: Cell-autonomous and non-cell-autonomous effects of TRIM72 on ALS disease progression

Xue Zhang, Ji He, Ning Song…·Yichang Jia

This study identifies TRIM72's protective role in ALS through both cell-autonomous and non-cell-autonomous mechanisms, showing its therapeutic potential in animal models and human patients.

3 citations

humanproteomics

𝕏 PostBluesky