Author

Julia V. Milne

The University of MelbourneORCID 0000-0002-8503-9304Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Nicholas James ClemonsiD· 3 shared Niko ThioiD· 3 shared Wayne Allen PhillipsiD· 3 shared Ebtihal H MustafaiD· 2 shared Kameng Wu· 2 shared Katherine Papastratos· 2 shared Maree Pechlivanis· 2 shared Sasha WittsiD· 2 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCancer Biology

Nicholas James ClemonsORCID

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3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJul 21, 2026

Mapping Functional Tumor Suppressor Networks in Esophageal Adenocarcinoma Using In Vivo CRISPR Screening and Perturb-sequencing

Ebtihal H Mustafa, Katherine Papastratos, Kameng Wu…·Nicholas James Clemons

The paper identifies tumor suppressors and their functional roles in esophageal adenocarcinoma progression using CRISPR screening and Perturb-seq.

DOI 10.64898/2026.07.19.739473

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Cancer BiologybioRxivPreprintMar 25, 2026

SMAD4 loss drives chromosomal instability during tumourigenesis via translational reprogramming

Julia V. Milne, Kameng Wu, Eric P. Kusnadi…·Nicholas James Clemons

This study shows SMAD4 loss reprograms translation to induce chromosomal instability, driving tumourigenesis, and identifies CDK11B-p58 as a key mediator.

DOI 10.64898/2026.03.23.712291

humanbulk RNA-seq

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Cancer BiologyCell Death and DiseasePeer-reviewedMar 1, 2024

Caspase-2 protects against ferroptotic cell death

Swati Dawar, Mariana Corrales Benitez, Yoon Pin Lim…·Nicholas James Clemons

This study shows caspase-2 protects mutant-p53 cancer cells from ferroptotic death by non-proteolytically interacting with proteins to limit GPX4 degradation.

23 citations

humanexperimental assay

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