Author

Jin Kuramochi

Tokyo Medical and Dental UniversityORCID 0000-0002-9078-0527Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 3 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 1 Approach:statistical model· 1 Organism:virus· 1

Frequent Co-Authors

Kei SatoiD· 3 shared Keiya Uriu· 3 shared Yu KakuiD· 3 shared Yusuke Kosugi· 3 shared Akifumi Takaori‐KondoiD· 2 shared Alfredo A. HinayiD· 2 shared Hiroyuki AsakuraiD· 2 shared Jumpei ItoiD· 2 shared

Activity by Year

2024-2024 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersMicrobiology

Kei SatoORCID

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3 papers

Recent·Most cited

MicrobiologyThe Lancet Infectious DiseasesPeer-reviewedMay 20, 2024

Virological characteristics of the SARS-CoV-2 KP.2 variant

Yu Kaku, Keiya Uriu, Yusuke Kosugi…·Kei Sato

This study investigates the virological properties of the SARS-CoV-2 KP.2 variant, finding it has higher viral fitness and immune resistance compared to JN.1, contributing to its rapid spread.

100 citationstop 1%

virusstatistical model

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MicrobiologyThe Lancet MicrobePeer-reviewedJan 11, 2024

Virological characteristics of the SARS-CoV-2 Omicron HK.3 variant harboring the "FLip" substitution

Yusuke Kosugi, Arnon Plianchaisuk, Olivia Putri…·Kei Sato

This study investigates the virological properties of the SARS-CoV-2 HK.3 variant, a FLip variant with substitutions S:L455F and S:F456L, to assess its growth advantage.

46 citationstop 1%

humanexperimental assay

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MicrobiologyThe Lancet Infectious DiseasesPeer-reviewedJan 8, 2024

Antiviral humoral immunity against SARS-CoV-2 Omicron subvariants induced by XBB.1.5 monovalent vaccine in infection-naive and XBB-infected individuals

Yusuke Kosugi, Yu Kaku, Alfredo A. Hinay…·Kei Sato

The paper assesses the humoral immune response to XBB.1.5 vaccine in infection-naive and XBB-infected individuals against various SARS-CoV-2 variants.

18 citationstop 4%

humanproteomics

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