Cell BiologybioRxivPreprint
Non-canonical ATR signalling via NBS1 phosphorylation propagates fork slowing from stressed to unperturbed nuclear regions
Jana Krietsch, Ilaria Ceppi, William J. Comstock…·Massimo Lopes
DOI 10.64898/2026.07.09.737522
Author
Diego Dibitetto
Mario Negri Institute for Pharmacological ResearchORCID 0000-0002-9549-5258Unclaimed ORCID profile · follow to get notified of new preprints
Biology Disciplines
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2022-2026 · peak 1/yrCo-Author Collaboration Network
10 top collaborators · 33 mutual co-author linksFocal author (center)Selected co-authorORCID co-authorUnlinked name
Curved chords = mutual co-authorship4 papers
Recent·Most cited
- Molecular BiologyNucleic Acids ResearchPeer-reviewed
The PIN1-p38-CtIP signaling axis protects stalled replication forks from deleterious degradation
Francesca Vivalda, Marco Gatti, Letizia Manfredi…·Alessandro A. Sartori
This study identifies the PIN1-p38-CtIP signaling pathway as essential for protecting stalled replication forks from degradation, independent of its role in homologous recombination.
3 citations - BiochemistryJournal of Biological ChemistryPeer-reviewed
In-Depth Mapping of DNA-PKcs Signaling Uncovers Conserved Features of Its Kinase Specificity
Shannon Marshall, M.V.A.S. Navarro, Carolline F.R. Ascenҫão…·Marcus Bustamante Smolka
This study identifies a novel phosphorylation motif used by DNA-PKcs, revealing its role in regulating splicing factors and transcriptional regulators across species.
- Molecular BiologybioRxivPreprint
MDC1 counteracts restrained replication fork restart and its loss causes chemoresistance in BRCA1/2-deficient mammary tumors
Martin Liptay, Joana S. Barbosa, Ewa Gogola…·Sven Rottenberg
This study identifies MDC1's role in replication fork restart and shows its loss leads to chemoresistance in BRCA1/2-deficient mammary tumors.
6 citations