Martin Kollmar

The paper uses proteogenomics to assess CUG codon mistranslation in Candida albicans and finds no evidence of leucine mistranslation, instead showing ribosomal-level ambiguity and no correlation with pathogenicity.

The paper evaluates coiled-coil prediction tools using PDB data, finding significant variation in predictions and limited applicability for large datasets.

The paper identifies the origin of a novel exocytic transport mechanism in early animal evolution, tracing SPIRE function to choanoflagellates and showing its role in coordinating actin/myosin forces for cellular communication.

The paper estimates the extent and biological role of mutually exclusive splicing in humans by identifying and validating over 855 MXEs, including novel exons, and links them to disease pathology.

The paper identifies a novel sense codon reassignment in Pachysolen tannophilus, where CUG is translated as alanine instead of leucine, and proposes a tRNA loss-driven mechanism for this genetic code alteration.