Author

Eva Maria Putz

St. Anna Children's Cancer Research InstituteORCID 0000-0002-9990-4477Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Synthetic Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:drug response / drug discovery· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:protein design· 1

Frequent Co-Authors

Dominik EmmingeriD· 3 shared Hayeon BaikiD· 3 shared Manfred LehneriD· 3 shared Michelle C. BuriiD· 3 shared Álvaro Muñoz‐LópeziD· 2 shared Benjamin SalzeriD· 2 shared Charlotte U. ZajciD· 2 shared Elise SylvanderiD· 2 shared

Activity by Year

2024-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersSynthetic Biology

Dominik EmmingerORCID

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3 papers

Recent·Most cited

Synthetic BiologybioRxivPreprintSep 9, 2025

MiniCARbids: Minimalistic human binding domains specifically tailored to CAR T applications

Magdalena Teufl, Julia Mayer, Kerstin E. Holzer…·Michael W. Traxlmayr

The paper introduces miniCARbids, small human-derived CAR binding domains that show high anti-tumor potency in CAR T cells, outperforming traditional scFvs in some cases.

1 citation

humanprotein sequence

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Synthetic BiologybioRxivPreprintJun 13, 2025

Caffeine-regulated molecular switches for functional control of CAR T cells in vivo

Elise Sylvander, Benjamin Salzer, Dominik Emminger…·Michael W. Traxlmayr

This paper develops caffeine-responsive switches for CAR T cells, showing they can be controlled by caffeine and effectively clear tumors in mice.

3 citations

mouseexperimental assay

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Cancer BiologyCancer Immunology ResearchPeer-reviewedDec 6, 2024

Natural killer cell cytotoxicity shapes the clonal evolution of B cell leukaemia

Michelle C. Buri, Mohamed R. Shoeb, Aleksandr Bykov…·Eva Maria Putz

This study shows NK cells influence all phases of cancer immunoediting in B-ALL, promoting resistant clones through selective pressure and identifying LY6E as a marker of poor prognosis.

4 citations

mousehuman

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