Author

Elizabeth R. Woodruff

University of Colorado AnschutzORCID 0000-0003-0237-7453Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

Benjamin Guy BitleriD· 3 shared Daniel Núñez-AvellanedaiD· 2 shared Fabian R. VillagómeziD· 2 shared Julie LangiD· 2 shared Margaret Cobb NevilleiD· 2 shared Patricia G. WebbiD· 2 shared Bogdan ConradiD· 1 shared Courtney A. BaileyiD· 1 shared

Activity by Year

2024-2024 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersCancer Biology

Benjamin Guy BitlerORCID

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3 papers

Recent·Most cited

Cancer BiologyCancer Research CommunicationsPeer-reviewedDec 3, 2024

Claudin-4 remodeling of nucleus-cell cycle crosstalk maintains ovarian tumor genome stability and drives resistance to genomic instability-inducing agents.

Fabian R. Villagómez, Julie Lang, Daniel Núñez-Avellaneda…·Benjamin Guy Bitler

This paper shows that claudin-4 remodels nucleus-cell cycle interactions to maintain genome stability and resist genomic instability-inducing agents in ovarian cancer.

6 citations

humanexperimental assay

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Cancer BiologybioRxivPreprintSep 19, 2024

Ablation of hematopoietic stem cell derived adipocytes reduces tumor burden in syngeneic mouse models of high-grade serous carcinoma

Elizabeth R. Woodruff, Courtney A. Bailey, Francis To…·Zachary L. Watson

This study shows that ablating hematopoietic stem cell-derived adipocytes reduces tumor burden in mouse models of high-grade serous carcinoma through metabolic and immune mechanisms.

DOI 10.1101/2024.09.19.613924

mousebulk RNA-seq

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Cancer BiologyCancer Research CommunicationsPeer-reviewedJun 13, 2024

Claudin-4 modulates autophagy via SLC1A5/LAT1 as a tolerance mechanism for genomic instability in ovarian cancer.

Fabian R. Villagómez, Julie Lang, Fredrick Joseph Rosario…·Benjamin Guy Bitler

This study shows claudin-4 promotes genomic instability tolerance in ovarian cancer by linking cell junctions to autophagy via SLC1A5/LAT1, and targeting it enhances PARPi efficacy.

10 citations

humanproteomics

𝕏 PostBluesky