Julie Jameson

The paper identifies TNFR2-expressing epidermal γδ T cells with an IL-17 gene signature in psoriasis, showing TNFR2's role in skewing these cells toward a Tγδ17 phenotype.

This study shows that tick extracellular vesicles alter keratinocyte function in the epidermis by inhibiting proliferation and wound closure, linked to phosphoinositide 3-kinase, KGF-1, and TGF-β.

This study shows that γδ T cells interact with NPY1R+ neurons in skin during wound repair, and obesity impairs these interactions, suggesting a neuroimmune disruption in wound healing.

This study investigates how obesity affects the CCR6-CCL20 axis and IL-17A production in epidermal γδ T cells during wound healing and psoriasis in mice.