Signe Lolle

The paper uses a human airway model to study co-infection dynamics between Staphylococcus aureus and Pseudomonas aeruginosa, revealing strain-specific traits and secretion systems that influence microbial competition.

This paper develops a dual-cell air-liquid interface model to study epithelial-immune interactions during airway infection, finding that macrophages modulate inflammation and bacterial distribution.

The paper investigates infection dynamics and virulence of Pseudomonas aeruginosa in human airway epithelial models, identifying four infection clusters based on strain-specific virulence and host response.

This study shows that mexZ mutations in Pseudomonas aeruginosa lead to antibiotic tolerance and altered colonization in human airway models through overexpression of lecA.