Stephanie D. Nofal

This study shows that phosphorylation of Plasmodium falciparum MyoA at Ser19 dynamically regulates motor speed and is essential for asexual replication.

The paper shows that febrile heat stress increases cytoadhesion in Plasmodium falciparum-infected red blood cells by enhancing surface protein trafficking, including PfEMP1 and PSAC.

The paper identifies a beta-hairpin motif in kinesin-2 that regulates motility by sequestering motor domains, revealing its role in activation and coordination with dynein.

This study investigates the evolution and inhibition of FIKK kinases in P. falciparum, showing they can be inhibited by a single compound.

The paper investigates the roles of CDC50 orthologues in Plasmodium falciparum, showing CDC50B modulates merozoite egress via GC interaction while CDC50C is essential for trophozoite maturation and haemoglobin processing.

This study shows that modifying the AP-2 adaptin complex in Plasmodium falciparum reduces susceptibility to artemisinin, suggesting alternative mechanisms of drug resistance beyond pfkelch13 mutations.