Krishna Rao Maddipati

This study shows that specific long-chain polyunsaturated fatty acids (LC-PUFAs) reduce CRC-induced muscle wasting in vitro by activating 15-LOX-dependent pathways and producing specialized pro-resolving lipid mediators.

This study shows ATG2A uses bridge-like lipid transport to regulate lipid droplet accumulation by efficiently moving phospholipids between membranes.

This study shows that 12/15-LOX is essential for inflammation resolution and skeletal muscle regeneration by regulating lipid mediators and myogenic progenitor cell function.