Author

Eva Rath

Technical University of MunichORCID 0000-0003-1910-1162Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Amira MetwalyiD· 2 shared Dirk HalleriD· 2 shared Doriane AguannoiD· 2 shared Elisabeth UrbaueriD· 2 shared Bärbel StecheriD· 1 shared David Gómez‐VarelaiD· 1 shared Dieter SauriD· 1 shared Feng XianiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersMolecular Biology

Amira MetwalyORCID

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2 papers

Recent·Most cited

Molecular BiologyCellular and Molecular Gastroenterology and HepatologyPeer-reviewedOct 4, 2025

Reduced intestinal GLP-1+ cell numbers are associated with an inflammation-related epithelial metabolic signature

Elisabeth Urbauer, Doriane Aguanno, Katharina Kuellmer…·Eva Rath

The paper shows reduced GLP-1+ cell numbers in inflamed intestinal tissues of mice and CD patients, linked to an inflammatory metabolic signature affecting epithelial differentiation.

4 citations

mousebulk RNA-seq

𝕏 PostBluesky

MicrobiologyCell Host & MicrobePeer-reviewedJul 15, 2024

Mitochondrial perturbation in the intestine causes microbiota-dependent injury and gene signatures discriminative of inflammatory disease

Elisabeth Urbauer, Doriane Aguanno, Nora Mindermann…·Dirk Haller

The paper shows mitochondrial dysfunction in intestinal epithelium leads to microbiota-dependent injury and identifies gene signatures that distinguish active from inactive inflammation in IBD.

60 citationstop 2%

mousebulk RNA-seq

𝕏 PostBluesky