Angel L Corbi

This study shows AhR restricts anti-inflammatory and pro-fibrotic traits in monocyte-derived macrophages by repressing key genes and limiting IL-10 signaling.

This study identifies how MAFB and MAF work together to shape human anti-inflammatory macrophage identity through IL-10 autocrine signaling and distinct functional roles.

This study shows that modulating GSK3 activity reprograms GM-CSF-dependent macrophages to exhibit features of M-CSF-dependent macrophages and interstitial macrophages.

The paper investigates resistance to JAK inhibitors in UC by analyzing single-cell RNA data from responders and non-responders, identifying hyperactivation of myeloid cells as a key mechanism.

This paper shows that the 5-HT2B agonist BW723C86 alters macrophage gene profiles via AhR and impairs osteoclast differentiation.