George W. Liechti

This study characterizes AmiA_Ct, a peptidoglycan amidase in Chlamydia trachomatis, showing its role in degrading peptidoglycan during cell division and supporting normal growth and development.

This study shows Chlamydia trachomatis delays NOD2 signaling until late in its developmental cycle, possibly to avoid immune detection, and suggests PG degradation reduces NOD2 ligand generation.

This study evaluates how Chlamydia trachomatis genomic DNA is recognized by TLR9 during infection and persistence, showing altered signaling during persistence.

This study compares gastric inflammation and metaplasia induced by Helicobacter pylori or Helicobacter felis in mice, finding that HF causes more severe inflammation and atrophy than HP.