Author

Anita K. Dunbier

University of OtagoORCID 0000-0003-4621-7419Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Approach:simulation· 1

Frequent Co-Authors

Michael A. BlackiD· 2 shared Conor McGuinnessiD· 1 shared Evangelia K. PapachristouiD· 1 shared Hamish D. McMillaniD· 1 shared Jason S. CarrolliD· 1 shared Jody HazlettiD· 1 shared Peter D. MaceiD· 1 shared Soleilmane OmarjeeiD· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

8 top collaborators · 22 mutual co-author links

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2 shared papersGenomics

Michael A. BlackORCID

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2 papers

Recent·Most cited

GenomicsCancer MedicinePeer-reviewedNov 17, 2023

Restriction site associated DNA sequencing for tumour mutation burden estimation and mutation signature analysis

Conor McGuinness, Michael A. Black, Anita K. Dunbier

This study shows that RADseq can be a cost-effective and accurate method for estimating tumour mutation burden and analyzing mutation signatures compared to existing methods.

3 citations

mousewhole-genome sequencing

𝕏 PostBluesky

Cancer BiologybioRxivPreprintJul 6, 2023

TRIB1 modulates transcriptional programming in breast cancer cells to regulate cell proliferation

Hamish D. McMillan, Evangelia K. Papachristou, Jody Hazlett…·Anita K. Dunbier

This study shows TRIB1 promotes breast cancer cell proliferation by regulating MYC expression and cell cycle progression through interactions with β-catenin and ER-driven transcription.

1 citation

humanbulk RNA-seq

𝕏 PostBluesky