Author

Maximilian Merten

Goethe University FrankfurtORCID 0009-0001-4793-7146Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Anita TamiatoiD· 2 shared Ariane FischeriD· 2 shared David JohniD· 2 shared Guillermo LuxániD· 2 shared Leah Rebecca Vanicek· 2 shared Stefanie DimmeleriD· 2 shared Badder KattihiD· 1 shared Bianca Schumacher· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersCell Biology

Anita TamiatoORCID

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2 papers

Recent·Most cited

Cell BiologyFEBS LettersPeer-reviewedMay 1, 2025

STAT3 expression is reduced in cardiac pericytes in HFpEF and its loss reduces cellular adhesion and induces pericyte senescence

Leah Rebecca Vanicek, Ariane Fischer, Mariano Ruz Jurado…·Guillermo Luxán

The paper identifies reduced STAT3 expression in cardiac pericytes in HFpEF and shows its loss leads to senescence and impaired adhesion.

4 citations

𝕏 PostBluesky

PhysiologyCirculation ResearchPeer-reviewedApr 2, 2024

Age-dependent RGS5 loss in pericytes induces cardiac dysfunction and fibrosis in the heart

Anita Tamiato, Lukas Tombor, Ariane Fischer…·Guillermo Luxán

This study shows that loss of RGS5 in pericytes with age leads to cardiac dysfunction and fibrosis by altering pericyte function and activating fibroblasts.

38 citationstop 1%

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