Author

Ryota Nakazato

Hiroshima UniversityORCID 0009-0006-5446-1189Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Faryal IjaziD· 2 shared Koji IkegamiiD· 2 shared H. OtaniiD· 1 shared Kanae Koike· 1 shared Kenichiro UemuraiD· 1 shared Kenji SHIRAKAWAiD· 1 shared Shinya TakahashiiD· 1 shared Tetsuhiro HaraiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 20 mutual co-author links

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2 shared papersCell Biology

Faryal IjazORCID

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2 papers

Recent·Most cited

Cell BiologyCell Structure and FunctionPeer-reviewedJan 1, 2026

Enhanced release of ciliary extracellular vesicles suppresses cell migration and promotes cell aggregation

Tetsuhiro Hara, Ryota Nakazato, Kenji SHIRAKAWA…·Koji Ikegami

This study shows that enhanced release of ciliary extracellular vesicles from PANC-1 cells suppresses migration and promotes aggregation, linking cilia function to cancer behavior.

DOI 10.1247/csf.25152

humanproteomics

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Cell BiologyJournal of Cell SciencePeer-reviewedJul 26, 2024

Excessive F-Actin and Microtubule Formation Mediates Primary Cilia Shortening and Loss in Response to Increased Extracellular Osmotic Pressure

H. Otani, Ryota Nakazato, Faryal Ijaz…·Koji Ikegami

This study shows that hyperosmotic shock causes reversible primary cilia shortening and loss in mouse cells, mediated by excessive microtubule and F-actin formation.

5 citations

mouseexperimental assay

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