Author

Kenichi Horisawa

Kyushu UniversityORCID 0009-0006-5944-0926Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Atsushi SuzukiiD· 2 shared Akihiko SakashitaiD· 1 shared Chisato KoderaiD· 1 shared Fumihito MiuraiD· 1 shared Hidetaka KatabuchiiD· 1 shared Hiromitsu Araki· 1 shared Hitoshi NiwaiD· 1 shared Kazumasa TakemotoiD· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

Atsushi SuzukiORCID

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2 papers

Recent·Most cited

Cell BiologyScientific ReportsPeer-reviewedDec 15, 2023

Transcription factor-mediated direct cellular reprogramming yields cell-type specific DNA methylation signature

Kenichi Horisawa, Shizuka Miura, Hiromitsu Araki…·Atsushi Suzuki

This study investigates DNA methylation changes during direct reprogramming of mouse embryonic fibroblasts to induced intestinal stem cells, showing similar methylation states between organoid-forming cells and minimal impact on gene expression.

2 citations

mouseDNA methylation

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Developmental BiologybioRxivPreprintJan 15, 2021

Meiosis-specific ZFP541 repressor complex promotes meiotic prophase exit during spermatogenesis

Yuki Takada-Horisawa, Chisato Kodera, Kazumasa Takemoto…·Kei‐ichiro Ishiguro

This study identifies a ZFP541-KCTD19 repressor complex that promotes meiotic prophase exit in spermatogenesis by suppressing transcriptional regulators and chromatin modifiers.

1 citation

mouseChIP-seq

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