Author
Boston Children's HospitalORCID 0009-0007-6946-0450Unclaimed ORCID profile · follow to get notified of new preprints
3 papers
Recent·Most cited
Victor Fattori, Fernanda Soares Rasquel-Oliveira, Soledad Ochoa…·Michael S. Rogers
This study identifies IL-33 as a key mediator in endometriosis pain and lesion formation through neuroimmune communication involving CGRP and macrophages.
Tiago H. Zaninelli, Victor Fattori, Olivia K. Heintz…·Michael S. Rogers
This study shows that blocking NGF-TrkA signaling reduces endometriosis-associated pain in mice, while blocking VEGF-VEGFR1 or BDNF-TrkB does not.
Victor Fattori, Tiago H. Zaninelli, Fernanda Soares Rasquel-Oliveira…·Michael S. Rogers
This study identifies CGRP/RAMP1 signaling as a driver of endometriosis pain and lesion growth, showing that blocking this pathway reduces pain and lesion size in mice.