Author

A. Finch

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

A. Pantazi· 1 shared A. Quintanilla· 1 shared E. Parasyraki· 1 shared F. L. Dix· 1 shared G. D. Love· 1 shared J. C. A. Read· 1 shared J. C. Acosta· 1 shared J. Patel· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

A. Pantazi

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Author profile →

2 papers

Recent·Most cited

Accommodation and wavelength: the effect of longitudinal chromatic aberration on the stimulus-response curve.

M. Fernandez-Alonso, A. Finch, G. D. Love, J. C. A. Read

This study examines how narrowband LED light affects accommodation and visual acuity, finding that accommodation compensates for longitudinal chromatic aberration with no difference in response variability between narrowband and broadband light.

bioRxiv · Jun 20, 2023 · PreprintPhysiology

Inhibition of the 60S ribosome biogenesis GTPase LSG1 causes endoplasmic reticular disruption and cellular senescence

A. Pantazi, A. Quintanilla, P. Hari, N. Tarrats, E. Parasyraki, F. L. Dix, J. Patel, T. Chandra, J. C. Acosta, A. Finch

This study shows that inhibiting LSG1, a GTPase involved in 60S ribosome biogenesis, induces cellular senescence through ER disruption and cholesterol biosynthesis upregulation, with potential therapeutic implications.

bioRxiv · Nov 8, 2018Cancer Biology