Author

A. Linge

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 3

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Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:clinical cohort / trial· 1

Frequent Co-Authors

A. Dubrovska· 2 shared C. Peitzsch· 2 shared F. Lohaus· 2 shared M. Baumann· 2 shared M. Krause· 2 shared U. Kahya· 2 shared V. Lukiyanchuk· 2 shared A. Abdollahi· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersCancer Biology

A. Dubrovska

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3 papers

Recent·Most cited

Blood-based detection of MMP11 as a marker of prostate cancer progression regulated by the ALDH1A1-TGF-β1 signaling mechanism

I. Gorodetska, V. Lukiyanchuk, M. Gawin, M. Sliusar, A. Linge, F. Lohaus, T. Hölscher, K. Erdmann, S. Fuessel, A. Borkowetz, M. Reardon, A. Choudhury, Y. Antonelli, A. Leal-Egana, A. S. Köseer, , A. Dubrovska

This study identifies MMP11 as a blood-based biomarker for prostate cancer progression, regulated by the ALDH1A1-TGF-β1 signaling pathway.

Journal of Experimental & Clinical Cancer Research · Mar 24, 2025Peer-reviewedCancer Biology

GLS-driven glutamine catabolism contributes to prostate cancer radiosensitivity by regulating the redox state, stemness and ATG5-mediated autophagy

A. Mukha, U. Kahya, A. Linge, O. Chen, S. Lock, V. Lukiyanchuk, S. Richter, T. C. Alves, M. Peitzsch, V. Telychko, S. Skvortsov, G. Negro, B. Aschenbrenner, I.-I. Skvortsova, P. Mirtschink, , A. Dubrovska

This study shows that glutamine catabolism via GLS regulates redox state, stemness, and autophagy in prostate cancer, affecting radiosensitivity and patient outcomes.

bioRxiv · Jan 7, 2021Cancer Biology

Subjugation of TGFβ Signaling by Human Papilloma Virus in Head and Neck Squamous Cell Carcinoma Shifts DNA Repair from Homologous Recombination to Alternative End-Joining

Q. Liu, L. Ma, T. Jones, L. Palomero, M. A. Pujana, H. Martinez-Ruiz, P. Ha, J. Murnane, I. Cuartas, J. Seoane, M. Baumann, A. Linge, M. H. Barcellos-Hoff

This study shows HPV-positive HNSCC cells lose TGFβ signaling, shifting DNA repair from homologous recombination to alternative end-joining, increasing PARP inhibitor sensitivity.

bioRxiv · Jun 26, 2018Cancer Biology