Author

M. Peitzsch

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3 Cell Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 6 Organism:mouse· 5 Research Focus:cell type annotation· 4 Assay:metabolomics· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:development & morphogenesis· 2 Research Focus:differential expression· 2 Organism:human· 2

Frequent Co-Authors

A. Witt· 2 shared B. Wielockx· 2 shared G. Eisenhofer· 2 shared H. Abdelmegeed· 2 shared I. Mateska· 2 shared P. Mirtschink· 2 shared T. Chavakis· 2 shared V. I. Alexaki· 2 shared

Activity by Year

2017-2022 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersImmunology

A. Witt

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Author profile →

6 papers

Recent·Most cited

Adrenal cortex size, homeostasis and tumorigenesis is regulated by gonadal hormones via androgen receptor/β-catenin signalling crosstalk.

R. Lyraki, A. Grabek, A. Tison, M. Peitzsch, N. Bechmann, S. Youssef, A. de Bruin, E. Bakker, F. Claessens, M.-C. Chaboissier, A. Schedl

The paper shows that gonadal hormones regulate adrenal cortex size and tumorigenesis via androgen receptor and β-catenin signaling crosstalk, leading to sex-specific outcomes in mice.

bioRxiv · Jun 23, 2022 · PreprintCancer Biology

Succinate mediates inflammation-induced adrenocortical dysfunction

I. Mateska, A. Witt, E. Hagag, A. Sinha, C. Yilmaz, E. Thanou, N. Sun, O. Kolliniati, M. Patschin, H. Abdelmegeed, H. Henneicke, W. Kanczkowski, B. Wielockx, C. Tsatsanis, A. Dahl, , V. I. Alexaki

This study identifies the IL-1β-DNMT1-SDHB-succinate axis as a mechanism by which inflammation impairs adrenal steroidogenesis.

bioRxiv · Apr 29, 2022Immunology

Innovative multidimensional models in a high-throughput-format for different cell types of endocrine origin

S. R. Bornstein, I. Shapiro, M. Malyukov, R. Züllig, E. Luca, E. Gelfgat, F. Beuschlein, S. Nölting, A. Berruti, S. Sigala, M. Peitzsch, C. Steenblock, B. Ludwig, P. Kugelmeier, C. Hantel

The paper develops standardized multidimensional models for endocrine cell types in a high-throughput format to study adrenal biology and disease.

bioRxiv · Jan 10, 2022Cancer Biology

GLS-driven glutamine catabolism contributes to prostate cancer radiosensitivity by regulating the redox state, stemness and ATG5-mediated autophagy

A. Mukha, U. Kahya, A. Linge, O. Chen, S. Lock, V. Lukiyanchuk, S. Richter, T. C. Alves, M. Peitzsch, V. Telychko, S. Skvortsov, G. Negro, B. Aschenbrenner, I.-I. Skvortsova, P. Mirtschink, , A. Dubrovska

This study shows that glutamine catabolism via GLS regulates redox state, stemness, and autophagy in prostate cancer, affecting radiosensitivity and patient outcomes.

bioRxiv · Jan 7, 2021Cancer Biology

Obesity-associated lipidomic remodeling of the adrenal gland indicates an important role of the FADS2-arachidonic acid axis in adrenocortical hormone production

A. Witt, P. Mirtschink, A. Palladini, I. Mateska, H. Abdelmegeed, M. Grzybek, B. Wielockx, M. Peitzsch, U. Coskun, T. Chavakis, V. I. Alexaki

This study identifies obesity-induced lipidomic changes in the adrenal gland, showing that the FADS2-arachidonic acid pathway regulates adrenocortical hormone production.

bioRxiv · Sep 4, 2020 · PreprintCell Biology

Compensation For Chronic Oxidative Stress In ALADIN Null Mice

R. Jühlen, M. Peitzsch, S. Gärtner, D. Landgraf, G. Eisenhofer, A. Huebner, K. Koehler

This study shows ALADIN null mice do not develop a triple A-like phenotype under chronic oxidative stress, indicating a compensatory mechanism in glutathione metabolism.

bioRxiv · Jul 17, 2017Cell Biology