Author

A. Marvin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Organism:mouse· 2 Organism:zebrafish· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

V. L. Bautch· 3 shared K. Quigley· 2 shared Z. Liu· 2 shared A. Gill· 1 shared A. Sharma· 1 shared D. B. Buglak· 1 shared D. L. Ruter· 1 shared E. J. Kidder· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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3 shared papersCell Biology

V. L. Bautch

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Author profile →

3 papers

Recent·Most cited

Endothelial cell flow-mediated quiescence is temporally regulated and utilizes the cell cycle inhibitor p27

N. Tanke, Z. Liu, M. T. Gore, P. Bougaran, M. B. Linares, A. Marvin, A. Sharma, M. Oatley, T. Yu, K. Quigley, S. Vest, J. G. Cook, V. L. Bautch

This study shows that endothelial cell quiescence under flow is temporally regulated and depends on p27, with arterial flow leading to deeper quiescence requiring HES1 and ID3.

bioRxiv · Jun 9, 2023Cell Biology

A defined clathrin-mediated trafficking pathway regulates sFLT1/VEGFR1 secretion from endothelial cells

K. Kinghorn, A. Gill, A. Marvin, R. Li, K. Quigley, F. le Noble, F. Mac Gabhann, V. L. Bautch

The paper identifies a clathrin-mediated trafficking pathway essential for sFLT1/VEGFR1 secretion from endothelial cells, revealing key components and their roles in vessel sprouting.

bioRxiv · Jan 27, 2023Cell Biology

SMAD6 Integrates Endothelial Cell Homeostatic Flow Responses Downstream of Notch

D. L. Ruter, Z. Liu, K. M. Ngo, S. X, A. Marvin, D. B. Buglak, E. J. Kidder, V. L. Bautch

This study shows SMAD6 is essential for endothelial cell alignment under laminar shear flow, downstream of Notch1, and its loss disrupts barrier function and gene expression.

bioRxiv · Jul 2, 2020Cell Biology