Author

A. P. Lightfoot

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:metabolomics· 1 Organism:mouse· 1

Frequent Co-Authors

A. Thoma· 2 shared R. G. Cooper· 2 shared G. Nye· 1 shared K. E. Earl· 1 shared K. Goljanek-Whysall· 1 shared M. Lyon· 1 shared N. Al-Shanti· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 15 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

A. Thoma

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cell BiologybioRxivJun 29, 2022

Major Histocompatibility Complex (MHC) I-induced Endoplasmic Reticulum (ER) Stress mediates the secretion of pro-inflammatory muscle-derived cytokines

A. Thoma, K. E. Earl, R. G. Cooper…·A. P. Lightfoot

This study shows that MHC I overexpression in muscle cells induces ER stress, leading to pro-inflammatory cytokine release, which is reduced by salubrinal.

DOI 10.1111/jcmm.17621

mouseproteomics

𝕏 PostBluesky

PhysiologybioRxivJun 10, 2020

Eukarion-134 attenuates endoplasmic reticulum stress-induced mitochondrial dysfunction in human skeletal muscle cells

A. Thoma, M. Lyon, N. Al-Shanti…·A. P. Lightfoot

This study shows that EUK-134 reduces ER stress-induced mitochondrial dysfunction in skeletal muscle cells by mitigating ROS generation and improving mitochondrial function.

DOI 10.3390/antiox9080710

humanproteomics

𝕏 PostBluesky