Author

A. Pathania

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Research Focus:drug response / drug discovery· 2 Assay:proteomics· 2 Assay:metabolomics· 1

Frequent Co-Authors

A. Gruss· 3 shared D. Halpern· 3 shared K. Gloux· 3 shared A. Solgadi· 2 shared G. Lamberet· 2 shared J. Anba-Mondoloni· 2 shared L. Dupont· 2 shared P. Trieu-Cuot· 2 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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3 shared papersMicrobiology

A. Gruss

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Prophages divert Staphylococcus aureus defenses against host lipids

B. Zhou, A. Pathania, D. Pant, D. Halpern, P. Gaudu, P. Trieu-Cuot, A. Dias-Leao, C. Pagot, A. Solgadi, A. Gruss, K. Gloux

This study shows that prophages in Staphylococcus aureus modulate lipid incorporation into bacterial membranes, affecting antibiotic resistance and infection dynamics.

bioRxiv · Jan 27, 2022 · PreprintMicrobiology

(p)ppGpp/GTP and malonyl-CoA modulate Staphylococcus aureus adaptation to FASII antibiotics and provide a basis for synergistic bi-therapy

A. Pathania, J. Anba-Mondoloni, M. Gominet, D. Halpern, J. Dairou, L. Dupont, G. Lamberet, P. Trieu-Cuot, K. Gloux, A. Gruss

This paper shows that (p)ppGpp and malonyl-CoA modulate S. aureus adaptation to FASII antibiotics, and that combining a (p)ppGpp inducer with an anti-FASII drug blocks bacterial outgrowth.

mBio · Feb 23, 2021Peer-reviewedMicrobiology

Permissive Fatty Acid Incorporation in Host Environments Promotes Staphylococcal Adaptation to FASII Antibiotics

G. Kenanian, C. Morvan, A. Weckel, A. Pathania, J. Anba-Mondoloni, D. Halpern, A. Solgadi, L. Dupont, C. Henry, C. Poyart, A. Fouet, G. Lamberet, K. Gloux, A. Gruss

This study shows that Staphylococcus aureus can adapt to FASII antibiotics by incorporating environmental fatty acids in host conditions, challenging the assumption of fatty acid stringency.

bioRxiv · Jun 17, 2019Microbiology