Author

A. Sanlaville

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

C. Caux· 2 shared J. Valladeau-Guilemond· 2 shared N. Bendriss-Vermare· 2 shared A. Puisieux· 1 shared A. Voissiere· 1 shared C. Brousse· 1 shared C. Perret· 1 shared D. Poujol· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCancer Biology

C. Caux

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintFeb 15, 2023

CD4 T cells and neutrophils contribute to epithelial-mesenchymal transition in breast cancer

A. Sanlaville, A. Voissiere, D. Poujol…·N. Bendriss-Vermare

The paper shows CD4 T cells and neutrophils promote EMT in breast cancer by analyzing murine tumor models and human cohorts.

DOI 10.1101/2023.02.15.528594

mousebulk RNA-seq

𝕏 PostBluesky

ImmunologybioRxivDec 23, 2020

cDC1 and interferons promote spontaneous CD4+ and CD8+ T cell protective responses to breast cancer

R. Mattiuz, C. Brousse, M. Ambrosini…·K. Crozat

This study shows that cDC1 and interferons are critical for CD4+ and CD8+ T cell responses against breast cancer, promoting infiltration and effector functions, and improving patient prognosis.

DOI 10.1002/cti2.1305

𝕏 PostBluesky