Author

Abigail Sondrini

University of Massachusetts Chan Medical School

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Kiera ClaytoniD· 2 shared Aaron ShulkiniD· 1 shared Alberto BosqueiD· 1 shared Alicja K. TrochaiD· 1 shared Alper KüçükuraliD· 1 shared Amanda Modica· 1 shared Fiachra HumphriesiD· 1 shared Komal Cheema· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersImmunology

Kiera ClaytonORCID

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2 papers

Recent·Most cited

ImmunologyThe Journal of ImmunologyPeer-reviewedJul 28, 2026

Non-canonical TLR signaling restricts cytosolic LPS detection.

Shriram Ramani, Sara E. Cahill, Matthew Finnegan…·Fiachra Humphries

This study identifies MARCO as a decoy LPS sensor regulated by non-canonical TLR signaling, which restricts inflammasome activation and prevents excessive inflammation.

DOI 10.1093/jimmun/vkag141.1471

mouseproteomics

𝕏 PostBluesky

ImmunologybioRxivPreprintApr 19, 2026

Loss of Vpr-driven TRAIL-R2 expression protects HIV-infected cells from non-canonical NK cell TRAIL attack

Paula E. Grasberger, Abigail Sondrini, Nicole C. Glidden…·Kiera Clayton

The paper shows HIV uses Vpr to increase TRAIL-R2 expression in CD4+ T cells, which enhances NK cell killing through non-canonical TRAIL signaling.

1 citation

humanproteomics

𝕏 PostBluesky