Author

B. D. Dynlacht

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Musacchio· 1 shared E. D'Amico· 1 shared G. Ciossani· 1 shared H. Itoh· 1 shared H. Nohira· 1 shared H. Sugihara· 1 shared M. Owa· 1 shared S. Wohlgemuth· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

A. Musacchio

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintApr 25, 2023

A mechanism that integrates microtubule motors of opposite polarity at the kinetochore corona

V. Cmentowski, E. D'Amico, G. Ciossani…·A. Musacchio

The paper identifies CENP-E's role in regulating DD kinetochore loading through the corona scaffold, ensuring coordinated bidirectional transport.

DOI 10.1101/2023.04.25.538277

humanproteomics

𝕏 PostBluesky

Cell BiologybioRxivPreprintMar 16, 2022

KIF24 controls the clustering of supernumerary centrosomes in pancreatic ductal adenocarcinoma cells

Y. Mashima, H. Nohira, H. Sugihara…·H. Itoh

This study shows that KIF24 depletion suppresses multipolar spindle formation and improves mitotic progression in PDAC cells with supernumerary centrosomes, independent of primary cilia.

DOI 10.1101/2022.03.16.484562

humanexperimental assay

𝕏 PostBluesky