Author

B. H. Schott

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

D. C. Ko· 2 shared A. L. Antonia· 1 shared A. P. Garza· 1 shared C. E. Anderson· 1 shared E. I. Buzas· 1 shared E. Pallinger· 1 shared E. Trahair· 1 shared I. R. Dunay· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersMicrobiology

D. C. Ko

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Author profile →

3 papers

Recent·Most cited

Initial and ongoing tobacco smoking elicits vascular damage and distinct inflammatory response linked to neurodegeneration

A. P. Garza, L. Morton, E. Pallinger, E. I. Buzas, S. Schreiber, B. H. Schott, I. R. Dunay

This study shows that tobacco smoking causes vascular damage and immune changes linked to neurodegeneration, even in asymptomatic individuals.

bioRxiv · Sep 27, 2022Immunology

Integration of the Salmonella Typhimurium methylome and transcriptome reveals DNA methylation and transcriptional regulation are largely decoupled under virulence-related conditions

J. Bourgeois, C. E. Anderson, L. Wang, J. L. Modliszewski, W. Chen, B. H. Schott, N. Devos, D. C. Ko

This study integrates DNA methylome and transcriptome data from Salmonella Typhimurium under different conditions, finding that DNA methylation is largely static and only rarely correlates with gene expression changes.

bioRxiv · Nov 11, 2021Microbiology

Pathogen evasion of chemokine response through suppression of CXCL10

A. L. Antonia, K. D. Gibbs, E. Trahair, K. J. Pittman, B. H. Schott, J. S. Smith, S. Rajagopal, J. W. Thompson, R. L. Reinhardt, D. C. Ko

This paper identifies a mechanism by which Leishmania major evades the immune system by cleaving CXCL10, a chemokine critical for T-cell recruitment, and shows similar strategies are used by other pathogens.

bioRxiv · Apr 29, 2019Microbiology