Author

B. S. Choudhary

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Shivashankar· 2 shared N. Chaudhary· 2 shared N. Verma· 2 shared S. Khanna· 2 shared S. Manna· 2 shared E. Jog· 1 shared J. Barr· 1 shared J. Dani· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCancer Biology

A. Shivashankar

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2 papers

Recent·Most cited

Cancer BiologybioRxivJul 17, 2023

EGFR-to-Src family tyrosine kinase switching in proliferating-DTP TNBC cells creates a hyperphosphorylation-dependent vulnerability to EGFR TKI

N. Chaudhary, B. S. Choudhary, A. Shivashankar…·N. Verma

This study identifies a hyper-phosphorylation state in drug-resistant TNBC cells caused by EGFR-to-Src kinase switching, making them vulnerable to EGFR TKI treatment.

DOI 10.1186/s12935-025-03691-4

humanproteomics

𝕏 PostBluesky

Cancer BiologybioRxivPreprintMay 18, 2023

GPX4-VIM equates a proliferating DTP state in TNBC subtypes with converged vulnerabilities to autophagy and glutathione inhibition.

N. Chaudhary, B. S. Choudhary, S. Patra…·N. Verma

The paper identifies a proliferating DTP state in TNBC subtypes that is vulnerable to autophagy and glutathione inhibition, linking GPX4-VIM expression to survival outcomes.

DOI 10.1101/2023.05.18.541287

humanproteomics

𝕏 PostBluesky