Author

B. Will

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

U. Steidl· 2 shared A. D'Alessandro· 1 shared A. Pellagatti· 1 shared A. Shastri· 1 shared A. Verma· 1 shared A. Wickrema· 1 shared A. Zintiridou· 1 shared B. Agianian· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCancer Biology

U. Steidl

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Activation of targetable inflammatory immune signaling is seen in Myelodysplastic Syndromes with SF3B1 mutations

G. Choudhary, A. Pellagatti, B. Agianian, M. Smith, T. D. Bhagat, S. Gordon-Mitchell, S. Pandey, N. Shah, S. Aluri, L. Schwartz, V. Steeples, R. Booher, M. Ramachandra, M. Samson, M. Carbajal, , A. Verma

This study identifies a longer, oncogenic IRAK4 isoform caused by SF3B1 mutations in MDS/AML, which is targetable by IRAK4 inhibition.

bioRxiv · Mar 9, 2022Cancer Biology

Cytoplasmic labile iron accumulates in aging stem cells perturbing a key rheostat for identity control

Y.-R. Kao, J. Chen, R. Kumari, M. Tatiparthy, Y. Ma, M. Aivalioti, A. Zintiridou, V. Thiruthuvanathan, J. Reisz, S. Stransky, S. Sidoli, U. Steidl, A. D'Alessandro, B. Will

This study shows that aging stem cells accumulate labile iron, disrupting a key regulatory pathway that maintains their identity and function.

bioRxiv · Aug 3, 2021 · PreprintCell Biology