Author

C. Bode

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Immunology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

D. Hoving· 1 shared I. V. Aramburu· 1 shared K. Klaus Stahl· 1 shared M. I. Temkin· 1 shared M. Ioannou· 1 shared M. Ralser· 1 shared N. M. De Vasconcelos· 1 shared Q. Wang· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

D. Hoving

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1 paper

Recent·Most cited

SIGNR1 promotes immune dysfunction in systemic candidiasis by modulating neutrophil lifespan via T cell-derived histones and G-CSF

M. Ioannou, D. Hoving, I. V. Aramburu, N. M. De Vasconcelos, M. I. Temkin, Q. Wang, S. Vernardis, V. Demichev, T.-D. Tsourouktsoglou, S. Boeing, R. Goldstone, S. David, K. Klaus Stahl, C. Bode, M. Ralser, V. Papagiannopoulos

This study identifies SIGNR1's role in promoting immune dysfunction during systemic candidiasis by modulating neutrophil lifespan through T cell-derived histones and G-CSF.

bioRxiv · Aug 9, 2021 · PreprintImmunology